oxygen toxicity

Progress in preventing acute oxygen toxicity with medicine in recent years

近年来药物预防急性氧中毒的研究进展

Fully developed blood vessels are not sensitive to oxygen toxicity .

充分发育的血管对氧中毒是不敏感的。

It can enhance the clinical effect and prevent the oxygen toxicity .

联合应用高压氧和旋磁场治疗脑卒中，不仅能够提高疗效，还能预防氧中毒的发生。

Aim : To further investigate the mechanism of pulmonary oxygen toxicity .

目的：进一步探索肺氧中毒的机理。

Changes of Serum Level of Cytokines in Mice after Acute Oxygen Toxicity

急性氧中毒小鼠血清细胞因子水平的变化

Effect of NF - κ B on Pulmonary Oxygen Toxicity

核因子κB在肺型氧中毒发病中的作用研究

Changes of platelet membrane glycoprotein expression in mice after acute oxygen toxicity

急性氧中毒小鼠血小板膜糖蛋白表达的变化

Effect of Macrophage Inflammatory Protein 2 on Pulmonary Oxygen Toxicity

巨噬细胞炎症蛋白-2在肺型氧中毒炎症发生中的作用研究

Oxygen toxicity is a complication of oxygen therapy .

氧中毒是氧疗的并发症。

Our investigation also showed that it is completely feasible to investigate acute oxygen toxicity using in vitro methods .

用体外实验方法研究氧中毒是完全可行的。

Effects of Cytochrome Oxidase Activity in Pulmonary Capillary of Mice Under Hyperbaric Oxygen Toxicity

高压氧中毒对肺毛细血管细胞色素氧化酶的影响高压氧中毒小鼠肺损伤及其机理

The Rapid Changes of Glutamic Acid Decarboxylase During Convulsive Type of Oxygen Toxicity

谷氨酸脱羧酶在急性脑型氧中毒中的快速变化

Experimental Study on Preventive Effect of Traditional Chinese Medicine Replenishing Recipe on Acute Oxygen Toxicity Caused by Hyperbaric Oxygen

中药补益方对高压氧致急性氧中毒预防作用的实验研究

Changes of macrophage inflammatory protein-2 mRNA expression in the lung tissue of pulmonary oxygen toxicity mice

肺型氧中毒小鼠肺组织巨噬细胞炎症蛋白-2mRNA表达的改变

Protection against Acute Oxygen Toxicity by Liposome-entrapped Superoxide Dismutase

脂质体包封超氧化物歧化酶预防急性氧中毒

Conclusion : PNS and GbE could effectively prevent acute oxygen toxicity , which were related to their antioxidant activities .

对过氧化氢酶和单胺氧化酶活性的影响则不显著。结论：三七总皂甙和银杏叶提取物可以有效预防急性氧中毒，其机制可能与它们的抗氧化活性有关。

Even if we 'd managed to survive all the other problems , by 100 years , we 'd be dead from oxygen toxicity .

即使我们能够克服其他所有困难，100年后我们还是会死于空气中毒。

The involvement of v β 3 integrin and complement activation provides a new area for finding therapeutic measure for pulmonary oxygen toxicity .

αvβ3整合素和补体系统的参与为寻找防治肺型氧中毒的有效手段提供了新的思路。

Objective To select the Chinese traditional drugs which could prevent acute oxygen toxicity and to inquire the relationship between their prophylactic effect and SOD activity .

目的选取能够有效地延缓急性氧惊厥发生的中药，探讨其预防作用及其与SOD活性的关系。

Conclusions HBO exposure could decrease GAD content in neurons , which may be an important factor correlative with the onset of acute oxygen toxicity .

结论HBO暴露导致神经元内GAD减少，可能是急性氧中毒发生的重要原因之一；

Studies on Preventive Effects of Sodium Ferulate , Chuanxiongzine , Panax notoginseng Saponins , Ginkgo biloba Extracts on Acute Oxygen Toxicity

阿魏酸钠、川芎嗪、三七总皂甙、银杏酮酯预防急性氧中毒作用的研究

Aim : To investigate the preventive effects of Panax notoginseng saponines ( PNS ) and Ginkgo biloba extracts ( GbE ) on acute oxygen toxicity and the possible mechanisms .

目的：研究三七总皂甙和银杏叶提取物对急性氧中毒的预防作用及其可能机制。

Results The expression of CD31 , CD61 and CD62p on platelet membrane in acute oxygen toxicity group mice significantly increased compared with normal control group mice ( respectively P < 0.05 , P < 0.05 , P < 0.01 ) .

结果急性氧中毒组小鼠血小板膜糖蛋白CD31和CD61的表达水平均高于正常对照组，有显著性差异（P均<0.05）；

Although researchers have devoted to studying oxygen toxicity for more than a century , the precise mechanisms of hyperoxia-induced seizures remain to be determined because of the difficulty of achieving the data from inside of the hyperbaric chamber and so on .

人们致力于研究氧惊厥的发生机制已有一个多世纪，但是由于包括舱内信号难以实时记录等技术原因，至今对其发生机制仍未能完全阐明。

The objective was to investigate the role and significance of cytokines in mice after acute oxygen toxicity . The serum level of IL-1 β, IL-6 and IL-10 in mice at the 1 h , 6 h and 12 h after acute oxygen toxicity was measured by enzyme-linked immuno-sorbent assay .

为探讨细胞因子在急性氧中毒中的作用和意义，用酶联免疫吸附法检测急性氧中毒后1、6和12h小鼠血清IL-1β、IL-6、IL-10的质量浓度。

But this direct contact often led to too much oxygen being absorbed and resulted in oxygen toxicity .

但这样的直接接触，常会导致血液的含氧量过高，造成氧中毒。

Objective : To observe the effects of hyperbaric oxygen on myeloperoxidase activity in the pulmonary tissue homogenate of mice with pulmonary oxygen toxicity and explore changes in lung tissue inflammation of mice with pulmonary oxygen toxicity .

目的：观察高压氧（HBO）暴露对小鼠肺组织匀浆中髓过氧化物酶（myeloperoxidase，MPO）活力的影响，探讨肺型氧中毒小鼠肺组织炎症的变化规律。

Results : oxygen concentration in premature infants was relatively high , and it was prone to get oxygen toxicity by taking oxygen with head net in various types .

[结果]采用不同型号的头罩吸氧其氧浓度高，容易发生氧中毒；

Oxygen concentration should be decreased while long time mechanical ventilation executed so as to reduce potential lung injury with oxygen toxicity .

结论：纯氧通气7h后观察到的肺超微结构损伤较多，吸入氧浓度34%则相对较少，建议长时间气管插管通气时，宜降低吸入氧浓度，尽可能减少潜在的氧毒性肺损伤。

Objective To investigate the effect of hyperbaric oxygen ( HBO ) exposure on the content of glutamic acid decarboxylase ( GAD ) in cultured neurons , and to offer some scientific evidences at cell level to elucidate the mechanism of acute oxygen toxicity .

目的研究高压氧（HBO）暴露对体外培养的神经元中谷氨酸脱羧酶（GAD）的影响，为从细胞水平阐明急性氧中毒的发生机理提供依据。